Session 1 — the foundational build the gaps stand on

Before the gap stress-tests, Session 1 built the foundation: five modules that emerge the cortical rhythm and its working memory, flip F1's false negative, add the axonal channel, derive the EM-wave physics, and probe cognition against the body — then run the full 21-module chain, whose open marker became Gap 1.

Sim 2's four gaps stand on a Session-1 foundation of five additive modules on the frozen kernel. Cortical emergence grows the rhythm from the measured FOXG1 γ (WM capacity ≈ 7.2 items, never told the band); the WM-holding module flips F1's false negative (pattern completion collapses 1.00 → 0.30 under disconnection, sign-stable); the axonal channel adds the second coupling and a leucotomy that spares perception; the EM-wave module shows the cortical population radiates at the speed of light yet couples as a sub-wavelength near-field (why Sim 1 is ephaptic); and the cognition/consciousness/body probes show access dissociates from arousal and the stream runs off the body (dreams). The full 21-module chain runs; its open marker — the field's causal role untested — became Gap 1.

The five foundational modules

Sim 2's gaps did not start from nothing. Session 1 built five additive modules on the frozen kernel — each grounded, each carrying new_tuned_constants = 0, each leaving the engine read-only — and those modules are the substrate the gap stress-tests then attacked. This page sets them out before the gaps.

Cortical emergence — the rhythm and its working memory

cortical_emergence grows the cortical rhythm from the measured FOXG1 γ (1.4737) through the frozen Population/FHN machinery, replacing a prior no-tuning violation (reading the atlas 40 Hz band and driving at it). The emerged theta/gamma working-memory capacity is ≈ 7.2 items — matching the engine's 6.125 and the Miller range — without the band being supplied. The grade is [V] for band identity and ratio, [L] for the cited band, and [O] for the absolute Hz. The honest residual is that the band is dominated by the inhibitory time constant τ, not the gene γ, so per-organ band assignment still rests on cited bands — [L], not [V].

Working-memory holding — flipping F1's false negative

cortical_wm_holding copies the frozen hippocampus micro-model as the cortical buffer (g = FOXG1), sized by the emerged working-memory capacity. Under the faithful disconnection lesion (severing the recurrent weights), pattern completion collapses monotonically and sign-stably (1.00 → 0.30), robust to the cell-gain convention. This flips F1's false negative: on the real working-memory substrate, holding is real and lesion-sensitive. The honest part is that the self-sustain read-out has a frac = 1.0 artifact (zero-field hysteresis), so the verdict rests on completion; and the autoassociator does not yet differentiate frontal from hippocampal (g does not enter the sign() dynamics). The grade is [V] holding-real, [O] frontal-specificity owed.

The axonal channel — a leucotomy that spares perception

frontal_axonal_channel adds the second physical coupling channel (the axonal connectome) Sim 1 omitted, with grounded relative strengths from cited tractography. The faithful leucotomy — sever the frontal↔thalamus axon but keep the field — spares perception across a κ sweep (sign-stable). The cortico-thalamic transmission “deficit”, by contrast, is circular (the read-out is the severed edge) and was demoted to a construction check. The grade is [L] perception-spared; the transmission deficit is not a finding. (Gap 3 develops this channel into the full O×W connectome.)

The EM wave — radiating at c, yet coupling as a near-field

frontal_em_wave emerges the cortical (FOXG1) population and computes its EM wave. The population radiates at the speed of light (front speed / c = 1.015; radiated energy > 0 → the measurable EEG gamma). But in tissue every brain rhythm is sub-wavelength — frontal gamma 40 Hz gives λ = 913 m, the hypothalamus at 2 Hz gives λ = 4082 m, and the brain is 0.15 m — so within the brain the coupling is quasi-static near-field. This is precisely why Sim 1 uses ephaptic coupling. The frontal gamma is the fastest major rhythm, hence the shortest wavelength and the most spatially-structured field; the slow hypothalamus is the pure near-field modulator. The grade is [V] Maxwell physics, [L] cited bands, [O] efficacy.

Cognition, consciousness, and the body — the three probes

cognition_consciousness_body operationalizes the three-layer framework. Probe A (the PCI analog) shows access dissociates from arousal: PCI is non-monotonic (it rises, peaks in the interior, then collapses) while activity rises monotonically — the vegetative/seizure signature. This dissociation is the basis the field-efficacy window (Gap 1) and the access window (Gap 4) build on; the honest correction is that the PCI peak sits at ~3× the measured coupling, so the real result is the dissociation, not a peak at measured. Probe B shows the stream of thought keeps strong momentum with the body decoupled (autocorr 0.95) versus coupled (0.55) — cognition runs off the body (dreams). The grade is [V] functional mechanisms, [O] felt quality.

The full chain and the open marker that became Gap 1

With the modules in place, the full chain M0–M20 runs (emerge_all, 21 modules). Its pervasive open marker is medium_efficacy_tested = 0.0 on M8–M19: the EM field's causal role is predicted, not shown. That open marker is exactly what Gap 1 sets out to fill — the field's causal window — and the four gaps follow from there.

Firewall (Axis-A). Every quantity on this page is a structural quantity of an in-silico model — a sign, a margin, an ordering — and is not a felt state, an experienced quality, or a level of consciousness (consciousness_claim = 0; the hard problem stays open, hard_problem_open = 1). These are model results, not validated neuroscience and not clinical guidance. efficacy = 0; not medical advice.