Carcinogen Dose Response and Relative Risk

A carcinogen is a sustained bias that lowers the R19 switch barrier, so malignant-crossing rate is Kramers over that barrier and relative risk is rate(dose)/rate(0). The lung-carcinoma curve starts exactly at RR(0)=1, has no threshold (half a pack-year already gives RR>1), and is convex, reaching ~18× at 100 pack-years. Forced endpoints [F]; shape [V]; absolute magnitude [O].

Barrier-lowering Beff = U(saddle)−U(basin) from the cubic s³−γs−h=0 gives RR = exp((B₀−Beff)/scale); RR(0)=1 [F], no threshold (slope √γ) [F], convex to ~18× at 100 pack-years [V]; absolute magnitude [O].

A carcinogen lowers the R19 barrier

Cell fate is the same R19 bistable switch. A carcinogen is a sustained aberrant bias hc that pushes toward the malignant basin and lowers the barrier the normal basin must climb. The malignant-crossing rate is Kramers/Arrhenius over that barrier, rate ∝ exp(−Beff/scale), and relative risk is RR(dose) = rate(dose)/rate(0).

The barrier is derived, not postulated: the three critical points are the real roots of the cubic s³ − γs − h = 0 (three reals inside |h| < h* = 2(γ/3)^1.5), and Beff = U(saddle) − U(basin). Two exact identities follow and are checked at import.

identityvaluegrade
Beff(h=0) equals substrate barrier γ²/40.5691[F]
barrier-drop slope at the origin equals √γ1.228[F]

Dose–response: monotone, convex, no threshold

Mapping cumulative tobacco exposure to bias gives a lung-carcinoma relative risk that starts exactly at RR(0) = 1, rises monotonically, and is convex (accelerating) — reaching 18.3× at 100 pack-years, the Doll & Peto order of magnitude.

There is no threshold: even half a pack-year already gives RR = 1.021 > 1. The low-dose slope is the forced quantity √γ, so any positive exposure raises risk — the linear-no-threshold shape, grade [V].

pack-yearsrelative riskln RR
01.00×0.000
51.23×0.205
101.50×0.405
202.20×0.788
303.15×1.148
404.41×1.484
506.03×1.796
608.02×2.082
8013.02×2.566
10018.32×2.908

Combination of carcinogens

Two carcinogens add their bias h. The model then predicts a combined risk that is 0.76× the product of the singles — sub-multiplicative at moderate dose, approaching multiplicative only in the low-dose limit. This is a falsifiable corollary; empirical agent-pair synergy varies.

What is open

The absolute relative-risk magnitude is grade [O]. Obstacle: the switch's effective noise temperature (scale) and the molecular dose-to-bias conversion are not fixed by substrate geometry and require an external population-hazard calibration — exactly as absolute organ size is open while size order is forced. The forced endpoints (RR(0)=1, no threshold) and the convex shape stand independently of that calibration.