A VP Theory volume · integrative capstone

Aging & Senescence

Aging is the slow loss of defense gain of every homeostatic setpoint, plus the accumulation of cells stuck in irreversible R19 attractors — and the dominant risk multiplier for every pathology kernel in the framework.

This volume derives aging on the jamming substrate: one declining-gain law gives both gradual drift and sudden failure (§3), senescence is an absorbing attractor (§4), telomere loss is reservoir depletion (§5), all ten hallmarks map cleanly (§6), and a ten-mammal test shows human aging genes are not special — the longevity switch is TP53 copy number, not promoter γ (§9).

human aging genes: not special (|z|<1)TP53 CV = 3.5% across 4–211 yrlongevity switch = copy number, not γage risk-multiplier 40→80 = 52.3×shared aging rate = 0.889

This package is derived from the VP Theory jamming branch → VP Physics, and it inherits node identities from the DNA blueprint. It re-emerges no organs owned elsewhere; it adds the temporal decline dynamics and owns the aging risk-multiplier seam for the rest of the framework.

Contents

Every quantity is reproduced deterministically (SEED 19, 2×sha256 identical) and graded honestly — [V] verified, [L] cited anchor, [O] open with a stated obstacle (§11). DOI is assigned on publication.

Cross-package connections

Builds on / references

Cited by

Mesh projected from registry/seam_edges.csv · machine-readable at seams.json